Neuromancer49

joined 1 year ago
[–] Neuromancer49@midwest.social 4 points 6 months ago (2 children)

Actually, we've got some pretty sophisticated models of neurons. https://en.wikipedia.org/wiki/Blue_Brain_Project?wprov=sfla1

See my other comment for an example of how little we truly understand about neurons.

[–] Neuromancer49@midwest.social 2 points 6 months ago (2 children)

Even assuming we can model the same number of (simple machine learning model) neurons, it's the connections that matter. The number of possible connections in the human brain is literally greater than the number of atoms in the universe.

[–] Neuromancer49@midwest.social 6 points 6 months ago* (last edited 6 months ago)

It's not a terrible idea by any means. It's pretty hard to do, though. Check out the Blue Brain Project. https://en.wikipedia.org/wiki/Blue_Brain_Project?wprov=sfla1

ETA: not to mention the brain is a heck of a lot more than a collection of neurons. Other commenters pointed out how we just discovered a new kind of brain cell - the brain is filled with so many different types of neurons (e.g. pyramidal, Purkinje, dopamine-based, myelinated, unmyelinated, internet Ron's, etc.). Then there's an entire class of "neuron support" cells called neuralgia. This includes oligodendrocytes (and Schwann cells), microglia, satellite cells, and most importantly, astrocytes. These star-shaped cells can have a huge impact on how neurons communicate by uptaking neurotransmitters and other mechanisms.

Here's more info: https://en.wikipedia.org/wiki/Tripartite_synapse?wprov=sfla1

[–] Neuromancer49@midwest.social 38 points 6 months ago (1 children)

Heck, we barely know how neurons work. Sure, we've got the important stuff down like action potentials and ion channels, but there's all sorts of stuff we don't fully understand yet. For example, we know the huntingtin protein is critical to neuron growth (maybe for axons?), and we know if the gene has too many mutations it causes Huntington's disease. But we don't know why huntingtin is essential, or how it actually effects neuron growth. We just know that cells die without it, or when it is misformed.

Now, take that uncertainty and multiply it by the sheer number of genes and proteins we haven't fully figured out and baby, you've got a stew going.

[–] Neuromancer49@midwest.social 3 points 7 months ago

Time to reboot and replay!!!

[–] Neuromancer49@midwest.social 4 points 7 months ago (3 children)

My understanding was you needed to have a second copy to 100% - I was always missing a specific mission item. So I never completed the game.

[–] Neuromancer49@midwest.social 16 points 7 months ago (5 children)

I loved FFTA so much, and did not care as much for FFTA2 on DS. Easily sunk 1000 hours into my GBA copy.

[–] Neuromancer49@midwest.social 12 points 8 months ago

siRNA and miRNA: Are we a joke to you?

[–] Neuromancer49@midwest.social 5 points 9 months ago (1 children)

Honestly, I started with an Ender 3 for less than $200 and it's just fine. You'll have to calibrate and fight with it for a bit, but once you get it running it's just fine.

[–] Neuromancer49@midwest.social 10 points 10 months ago (1 children)

I'm 75% sure bone meal doesn't work on sugar cane. Scaling up is the only option.

[–] Neuromancer49@midwest.social 2 points 11 months ago

I continue to feed my X4 addiction. Picked up the DLC during the Thanksgiving sale. I'm on my second play, about 35 hours in. Realized I've made a lot of mistakes, but that's half the fun.

Fair point. But, I think the headline was a but sensationalized. Or, I'm just bad at reading, which is possible.

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